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NABP FPGEE Exam - Topic 7 Question 65 Discussion

Actual exam question for NABP's FPGEE exam
Question #: 65
Topic #: 7
[All FPGEE Questions]

Mechanism of resistance of dicloxacillin is:

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Suggested Answer: D

by Merilyn at Jul 08, 2023, 01:42 PM

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Lindsey
3 months ago
I thought mutations in M protein were more common.
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Ernie
3 months ago
Antibodies from strep infection? That sounds off.
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Howard
4 months ago
Wait, are we sure it's not just decreased drug uptake?
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Robt
4 months ago
Definitely agree, PBPs are key here too.
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Sonia
4 months ago
It's all about the beta-lactamase!
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Rodolfo
4 months ago
I don't remember much about E, but antibodies from streptococcal infections seem unrelated to dicloxacillin resistance. I lean towards C or D.
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Sang
4 months ago
I recall a practice question about resistance mechanisms, and I think decreased drug uptake was mentioned, so maybe A is a possibility too?
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Quentin
5 months ago
I'm not entirely sure, but I feel like it could also be related to D, the penicillin binding proteins. We discussed that in class.
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Clarinda
5 months ago
I think the answer might be C, beta-lactamase, since I remember it being a common resistance mechanism for penicillins.
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Gerald
5 months ago
Ooh, this is an interesting one! I like the idea of using a custom object to store the questions and answers, and then leveraging LWC to display them dynamically. That could give us a lot of flexibility and control over the user experience.
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Juliana
5 months ago
Hmm, this looks like a tricky one. I'll need to think carefully about the diagnostic tools used with Backup Exec.
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Remedios
5 months ago
Okay, let me think this through. GV is expanding into a new product category (leather shoes) in a new market (country P), so I believe the correct answer is related diversification.
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William
9 months ago
I'm just going to go with C) Beta-lactamase and hope for the best. If this is wrong, I'll be dicloxappointed.
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Lashaun
10 months ago
E) Antibodies resulting from streptococcal infection? Really? That's just silly. Maybe the exam writers have a twisted sense of humor.
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Donette
8 months ago
D) Penicillin binding proteins (PBPs)
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Marti
9 months ago
C) Beta-lactamase
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Demetra
9 months ago
A) Decreased drug uptake by bacteria
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Gianna
10 months ago
This is a tricky one. I'm going to go with B) Mutations in M (matrix) protein. Sounds like something a sneaky bacteria would do.
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Melodie
10 months ago
Hmm, I'm not so sure. I was thinking it might be D) Penicillin binding proteins (PBPs). Isn't that how some bacteria become resistant?
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Cory
9 months ago
E) Antibodies resulting from streptococcal infection
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Mirta
9 months ago
C) Beta-lactamase
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Josefa
9 months ago
A) Decreased drug uptake by bacteria
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Chaya
10 months ago
I think it's definitely C) Beta-lactamase. That's the classic mechanism for penicillin resistance.
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Golda
8 months ago
I've heard that some bacteria can also develop mutations in M protein.
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Berry
8 months ago
True, PBPs can also play a role in resistance.
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Jodi
8 months ago
But I think it could also be D) Penicillin binding proteins (PBPs).
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Blair
8 months ago
I agree, Beta-lactamase is a common mechanism for resistance.
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Salome
8 months ago
It's interesting how bacteria can develop different mechanisms to resist antibiotics.
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Hollis
9 months ago
True, PBPs can definitely contribute to resistance as well.
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Sabra
9 months ago
I think it could also be D) Penicillin binding proteins (PBPs) playing a role.
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Roslyn
10 months ago
I agree, Beta-lactamase is a common mechanism for resistance.
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Oliva
10 months ago
I believe it's Penicillin binding proteins (PBPs) that are responsible for resistance to dicloxacillin.
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Daryl
10 months ago
I agree with Wenona, Beta-lactamase can break down dicloxacillin.
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Wenona
11 months ago
I think the mechanism of resistance of dicloxacillin is Beta-lactamase.
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Royal
11 months ago
Hmm, that makes sense too. Beta-lactamase can definitely break down dicloxacillin.
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Lavonna
11 months ago
I disagree, I believe it is because of beta-lactamase production by bacteria.
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Royal
11 months ago
I think the mechanism of resistance of dicloxacillin is due to mutations in M protein.
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